The hematopoietic transcription factor PU.1 regulates RANK gene expression in myeloid progenitors - ScienceDirect
GAPDH Antibody (1D4) (NB300-221): Novus Biologicals
A transcription factor regulatory circuit in differentiated pancreatic cells | PNAS
CCR2+CD103− intestinal dendritic cells develop from DC-committed precursors and induce interleukin-17 production by T cells | Mucosal Immunology
Th2 cell‐derived IL‐4/IL‐13 promote ILC2 accumulation in the lung by ILC2‐intrinsic STAT6 signaling in mice - Symowski - 2019 - European Journal of Immunology - Wiley Online Library
Bcl11a is essential for normal lymphoid development | Nature Immunology
Srf−/− ES cells display non‐cell‐autonomous impairment in mesodermal differentiation | The EMBO Journal
The Role of Kv1.2 Channel in Electrotaxis Cell Migration - Zhang - 2016 - Journal of Cellular Physiology - Wiley Online Library
E2A/HEB and Id3 Proteins Control the Sensitivity to Glucocorticoid-induced Apoptosis in Thymocytes by Regulating the SRG3 Expression* - Journal of Biological Chemistry
FGF21 modulates mitochondrial stress response in cardiomyocytes only under mild mitochondrial dysfunction | Science Advances
Th17 immunity in the colon is controlled by two novel subsets of colon-specific mononuclear phagocytes | bioRxiv
Biomedicines | Free Full-Text | Comparative Analysis of the Transcriptome, Proteome, and miRNA Profile of Kupffer Cells and Monocytes | HTML
miR-181a Modulation of ERK-MAPK Signaling Sustains DC-SIGN Expression and Limits Activation of Monocyte-Derived Dendritic Cells - ScienceDirect
Identification of suitable reference genes for gene expression studies in rat skeletal muscle following sciatic nerve crush injury
miR‐191 modulates B‐cell development and targets transcription factors E2A, Foxp1, and Egr1 - Blume - 2019 - European Journal of Immunology - Wiley Online Library
Neil2-null Mice Accumulate Oxidized DNA Bases in the Transcriptionally Active Sequences of the Genome and Are Susceptible to Innate Inflammation*♢ - Journal of Biological Chemistry
Lipid-Droplet Formation Drives Pathogenic Group 2 Innate Lymphoid Cells in Airway Inflammation - ScienceDirect
